70 N+S Experiments proved that fructose caused the development of insulin resistance in humans.

QUOTE from Dr Fung (emphasis mine): 

“At the liver, fructose is rapidly metabolized into glucose, lactose and glycogen. The body handles excess glucose consumption through several well-defined metabolic pathways, such as glycogen storage and de novo lipogenesis (creation of new fat). No such system is present for fructose. The more you eat, the more you metabolize. The bottom line is that excess fructose is changed into fat in the liver. High levels of fructose will cause fatty liver. Fatty liver is absolutely crucial to the development of insulin resistance in the liver. 

That fructose directly causes insulin resistance was discovered long ago. As far back as 1980, experiments proved that fructose (but not glucose) caused the development of insulin resistance in humans.16 Healthy subjects were given an extra 1000 calories per day of either glucose or fructose. The glucose group showed no change in insulin sensitivity. The fructose group, however, showed a 25 percent worsening of their insulin sensitivity—after just seven days!” [1]

NOTE (my commentary)

Text The bottom line is that excess fructose is changed into fat in the liver.

High levels of fructose will cause fatty liver.

Text

experiments proved that fructose (but not glucose) caused the development of insulin resistance in humans.

Text

STRATEGY:

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SOURCE – Footnotes:
[1]  “The Obesity Code: Unlocking the Secrets of Weight Loss” by Dr. Jason Fung, Timothy Noakes, page 164.

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Key: Fung01-16.70 Last Revision: 02/02/2021

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